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UMN Facial Weakness – Bilateral Innervation — SCE Neurology MCQ

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HardCerebrovascular DiseaseUMN Facial Weakness – Bilateral InnervationSCE Neurology

A right corticobulbar lesion causes left lower facial weakness with preserved forehead movement. A later independent lesion interrupts the left corticobulbar fibres above the facial nucleus. Under the classical corticobulbar model, which new voluntary facial pattern is most plausible?

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Correct answer: DBilateral lower facial weakness with additional loss of the remaining cortical drive to both foreheads

Each upper facial motor subnucleus receives bilateral corticobulbar input, whereas each lower facial subnucleus receives predominantly contralateral input. The first unilateral lesion therefore weakens the contralateral lower face but leaves upper facial movement supported by the other hemisphere. A second contralateral supranuclear lesion removes that remaining bilateral cortical contribution and also weakens the opposite lower face. This is a bilateral supranuclear pattern, not a unilateral nuclear or peripheral facial palsy.

Reference: Facial Nerve Anatomy and Clinical Applications. https://www.ncbi.nlm.nih.gov/books/NBK554569/