skip to main content

Baroreflex Activation Therapy — EECC MCQ

Instant feedback + full explanation. One question, done properly.

HardHypertension & Preventive CardiologyBaroreflex Activation TherapyEECC

A 65-year-old man with resistant hypertension (BP 158/95 on 4 drugs including spironolactone) asks about device-based treatments beyond renal denervation. What other device therapies have been investigated for resistant hypertension?

Educational content. Not a substitute for clinical judgement or local policy.

Reveal the answer and explanation

Correct answer: CBaroreflex activation therapy (BAT) — electrical stimulation of the carotid baroreceptors increases parasympathetic and decreases sympathetic outflow, reducing BP; the Rheos and Barostim neo trials showed significant BP reduction in resistant HTN, though adoption is limited by device cost and invasiveness

Device-based therapies for resistant hypertension are evolving: (1) Renal denervation: catheter-based ablation of renal sympathetic nerves (SPYRAL, RADIANCE trials — ~5-10 mmHg SBP reduction, Class IIb); (2) Baroreflex activation therapy (BAT): surgically implanted device electrically stimulates the carotid sinus baroreceptors, triggering the baroreflex arc → increased parasympathetic output, decreased sympathetic output → BP reduction. Rheos system (bilateral carotid stimulation) → Barostim neo (unilateral, simpler). Clinical evidence: significant BP reductions (20-30 mmHg SBP in some studies), but limited by: device cost, surgical implantation, battery life, and limited long-term data; (3) Arteriovenous anastomosis (ROX coupler): creates a controlled iliac AV fistula to reduce peripheral resistance — promising early results but limited adoption. The 2024 ESC HTN Guidelines position these as investigational/limited-availability options for truly refractory hypertension.

Reference: ESC (2024): Hypertension Guidelines