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ukmla 2026

Dermatophyte Fungal Infections (Tinea)

Superficial fungal infections of keratinised tissue caused by dermatophytes — classified by site as tinea corporis, pedis, capitis, cruris, unguium

Dermatologycommonacute
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Condition details
Dermatology
common
5 min read
reviewed 2026-04-05
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About This Page

This is a clinician-written, evidence-based summary aligned to the 2026 MLA Content Map. It is intended for medical students and junior doctors preparing for the UKMLA. Always cross-reference with NICE guidance, local protocols, and clinical judgement.

Key points

  • Named by site: corporis (body), pedis (feet), cruris (groin), capitis (scalp), unguium (nails)
  • Classic: annular erythematous scaly plaque with central clearing and advancing border
  • Skin scraping for KOH microscopy and fungal culture from advancing edge
  • Skin: topical terbinafine cream OD-BD for 1-2 weeks
  • Tinea capitis and unguium ALWAYS need ORAL antifungal — terbinafine or griseofulvin

Overview

Dermatophyte infections are caused by fungi (Trichophyton, Microsporum, Epidermophyton) that metabolise keratin. Named by site. Classic ringworm appearance: annular plaque with central clearing and advancing scaly border. Tinea capitis primarily affects children (Trichophyton tonsurans commonest in UK). Tinea pedis is the commonest portal of entry for lower limb cellulitis.

Epidemiology

Extremely common — tinea pedis affects 15-25% of population. Risk factors: warm/moist environments, communal bathing, diabetes, immunosuppression, occlusive footwear.

Clinical Features

Symptoms
Tinea corporis: annular scaly patches with central clearing
Tinea pedis: itchy, scaly skin between toes (4th/5th web space)
Tinea capitis: scalp scaling, alopecia patches, kerion
Tinea unguium: thickened, discoloured, dystrophic nails
Signs
Annular plaque with peripheral scale, advancing border, central clearing
Tinea pedis: interdigital maceration, moccasin pattern scaling
Tinea capitis: broken hairs, black dots, kerion (boggy inflammatory mass)
Onychomycosis: distal subungual discolouration, onycholysis, hyperkeratosis

Investigations

First-line
Skin scraping for mycologyFrom advancing edge — KOH microscopy (rapid) and fungal culture (4-6 weeks)
Nail clippingsConfirm fungal infection before starting oral antifungals
Second-line
Wood's lampMicrosporum fluoresces green. T. tonsurans does NOT
1
Skin (corporis, cruris, pedis) — topical
  • Terbinafine 1% cream OD-BD for 1-2 weeks (most effective)
  • OR clotrimazole 1% cream BD for 4 weeks
2
Tinea capitis — ORAL required
  • Topicals alone are INEFFECTIVE
  • Terbinafine 250 mg OD 4 weeks (Trichophyton) or griseofulvin (Microsporum, children)
  • Adjunctive antifungal shampoo (ketoconazole 2%)
3
Tinea unguium — ORAL required
  • Confirm with mycology BEFORE treatment
  • Terbinafine 250 mg OD: 6 weeks fingernails, 12 weeks toenails
  • Itraconazole pulse therapy as alternative

Complications

  • Cellulitis: Tinea pedis is commonest entry point for lower limb cellulitis
  • Kerion: Inflammatory tinea capitis — risk of scarring alopecia
  • Id reaction: Secondary eczematous eruption as immune response to distant fungal infection
UKMLA Exam Tips
  • 1Tinea capitis ALWAYS needs oral antifungal — topical alone insufficient
  • 2Tinea pedis is key predisposing factor for cellulitis — check between toes
  • 3Confirm nail infection with mycology BEFORE prescribing oral terbinafine
  • 4Kerion is NOT an abscess — do NOT incise. Treat with oral antifungal
  • 5Annular lesion + central clearing + advancing scaly border = ringworm

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regional clinical guidance

Dermatophyte Fungal Infections (Tinea): guidance by region

Recommendations, thresholds and pathways can differ. Open the page written for the jurisdiction you need.

Verified Sources & References

NICE CKS — Fungal skin infection
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