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mccqe1 clinical presentation

Gait Abnormality

Gait disturbance localises disease across cerebellar, sensory, vestibular, pyramidal, extrapyramidal, frontal and musculoskeletal systems — falls risk and acute ataxia drive urgency.

NeurologicalurgentMusculoskeletal & RheumatologicGeneral & Constitutional
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Presentation details
Neurological
urgent
spans 3 domains
9 min read
reviewed 2026-05-04
MCCobjective #35
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About This Page

This is a clinician-written, evidence-based guide aligned to the MCC Examination Objectives. It is structured by clinical presentation — the way the MCCQE tests and the way patients actually present. Management reflects current Canadian guidelines (CMA, CFPC, CPS). Always cross-reference with institutional protocols and clinical judgment.

Key points

  • Observe gait before touching the patient: base, stride, arm swing, turning, posture and need for aids.
  • Acute ataxia is cerebellar stroke/haemorrhage until proven otherwise.
  • Sensory ataxia worsens with eyes closed and has impaired vibration/proprioception.
  • Parkinsonian gait has shuffling, reduced arm swing and en bloc turning; NPH has magnetic gait with cognitive and urinary symptoms.
  • Gait problems require falls-risk assessment and medication review.

Approach to the Presentation

Classify gait by pattern and time course. Acute vertigo/ataxia needs central cause exclusion. Chronic gait disturbance often reflects multi-factorial disease: neuropathy, vision, vestibular dysfunction, Parkinsonism, arthritis, medications and cognition.
Differential Diagnosis
diagnosislikelihoodkey featuresdistinguishing test
Cerebellar stroke/haemorrhagemust-not-missAcute wide-based gait, dysmetria, nystagmus, vomiting, headache, inability to sit/standUrgent CT/MRI brain and vascular imaging
Cervical/thoracic myelopathymust-not-missSpastic gait, falls, hand clumsiness, hyperreflexia, sensory level, bladder symptomsMRI spine
Parkinson diseasecommonShuffling gait, reduced arm swing, stooped posture, freezing, rest tremor/bradykinesiaClinical
Sensory ataxia / neuropathycommonStomping gait, worse in dark, loss of vibration/proprioception, positive RombergB12/A1c and NCS if needed
Vestibular disordercommonVeering, vertigo, nausea, nystagmusVestibular exam/Dix-Hallpike
Musculoskeletal/antalgic gaitcommonPain-limited limp, hip/knee/foot pathologyMSK exam and targeted imaging
Medication/alcohol effectcommonSedatives, anticonvulsants, alcohol causing ataxia or fallsMedication review
Normal-pressure hydrocephalusless commonMagnetic gait, urinary urgency/incontinence, cognitive declineCT/MRI ventriculomegaly; specialist tap test
Frontal gait disorderless commonDifficulty initiating, broad-based short steps, vascular cognitive impairmentBrain imaging

Red Flags & Key History

Symptoms
Acute onset ataxia or inability to walk
Back/neck pain with spastic gait or bladder symptoms
Recurrent falls with injury
Cognitive decline plus urinary symptoms
Worse in dark
Signs
Truncal ataxia
Hyperreflexia/clonus/Babinski
Positive Romberg with proprioceptive loss
Shuffling with reduced arm swing

Approach to Investigation

First-line
Direct gait observationNormal walk, tandem, turns, chair rise, Romberg if safe.
Full neuro/MSK examPower, tone, reflexes, cerebellar signs, proprioception, vision, feet and joints.
Medication and falls reviewSedatives, antihypertensives, anticholinergics and alcohol.
Second-line
MRI/CT brainAcute ataxia, focal signs, NPH or central cause.
MRI spineMyelopathic signs or sensory level.
B12, A1c, TSH, CBCReversible gait contributors.
Specialist
Physio/OT/falls clinic/neurologyFalls risk, progressive neurological gait, Parkinsonism or suspected NPH/myelopathy.
1
Emergency gait syndromes
  • Activate stroke pathway for acute ataxia
  • Urgent MRI spine for myelopathy/cord features
2
Chronic gait/falls
  • Multifactorial falls assessment, medication reduction, vision/footwear review and strength/balance training
  • Treat underlying neuropathy, Parkinsonism, NPH or MSK pain
3
Rehabilitation
  • Physiotherapy, occupational therapy, gait aids and home safety interventions

Complications & Pitfalls

  • Not watching the gait: the pattern is often diagnostic.
  • Missing cerebellar stroke: acute inability to walk is central until proven otherwise.
  • Ignoring myelopathy: spastic gait plus hand clumsiness needs spine MRI.
  • Forgetting medications: sedatives and anticholinergics are common falls causes.
MCCQE1 Exam Tips
  • 1Acute ataxia = posterior circulation stroke until proven otherwise.
  • 2Positive Romberg = sensory or vestibular dependence, not cerebellar ataxia by itself.
  • 3NPH gait is the earliest and most treatment-responsive feature.
  • 4Parkinson gait has reduced arm swing and en bloc turning.
  • 5Myelopathy causes spastic gait and hyperreflexia.
  • 6Falls prevention is a management answer, not just diagnosis.

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Verified Sources & References

MCC Objective: Ataxia (gait)
Canadian guideline for Parkinson disease
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